Intestinal wall necrosis

Introduction

Introduction Intestinal wall necrosis is common in intestinal blockage. Because the intestinal contents such as mites, gallstones, feces or other foreign bodies block the intestinal lumen, it is called a simple mechanical intestinal obstruction. More common is that the aphids clump together and cause local intestinal fistulas and block the intestinal lumen. Most common in children, the incidence rate in rural areas is high. The clinical manifestations are paroxysmal abdominal pain and vomiting around the umbilicus, which may have a history of aphids or spit mites. The clinical manifestations are paroxysmal abdominal pain and vomiting around the umbilicus, which may have a history of aphids or spit mites. Generally, the abdominal distension is not significant, and there is no abdominal muscle tension. The abdomen often has a strip-like mass that can be deformed and displaced, and may become hard with the contraction of the intestinal tube, and the bowel sounds may be hyperthyroidized or normal.

Cause

Cause

Intestinal wall necrosis is common in intestinal blockage. Because the intestinal contents such as mites, gallstones, feces or other foreign bodies block the intestinal lumen, it is called a simple mechanical intestinal obstruction. More common is that the aphids clump together and cause local intestinal fistulas and block the intestinal lumen. Most common in children, the incidence rate in rural areas is high.

Examine

an examination

Related inspection

Gastrointestinal CT examination of gastrointestinal diseases by ultrasound examination of gastrointestinal dysfunction

First, physical examination

Taking a medical history gives us a first impression and revelation, and also guides us to a concept of the nature of the disease.

Second, laboratory inspection

Laboratory examinations must be summarized and analyzed based on objective data learned from medical history and physical examination, from which several diagnostic possibilities may be proposed, and further consideration should be given to those examinations to confirm the diagnosis. Such as: body temperature, white blood cell counts are more normal. On the abdominal x-ray film, in addition to the small intestine inflation or liquid level, sometimes the shadow of the worms in the intestinal cavity can be seen. Diagnosis is generally not difficult, but attention should be paid to the identification of intussusception. A small number of patients can be complicated by intestinal torsion or necrosis of the intestinal wall, and a large number of aphids enter the abdominal cavity to cause peritonitis.

Diagnosis

Differential diagnosis

Intestinal wall edema thickening: dermatomyositis has different degrees of dilatation and segmental changes, with low power and prolonged passage. Intestinal wall edema and thickening can be seen with the naked eye. Microscopic examination revealed multiple mucosal erosions, submucosal edema, muscular atrophy, and fibrosis with lymphatic and plasma cell infiltration. Intestinal submucosal to serosal small artery, venule intima thickening is prone to thrombosis and luminal occlusion.

Intestinal wall fibrosis: schistosomiasis granuloma in the ileocecal area is the venous oocysts that swell into the intestinal wall tissue around the blood vessels, especially the submucosa. Insect eggs cause leukocyte infiltration in the intestinal wall, pseudo nodules are formed, fibrous tissue is proliferated, late intestinal wall fibrosis is thickened, and mucosal proliferation forms granuloma.

Intestinal wall necrosis: It is a simple mechanical intestinal obstruction because it is blocked by intestinal contents such as mites, gallstones, feces or other foreign bodies. More common is that the aphids clump together and cause local intestinal fistulas and block the intestinal lumen. Most common in children, the incidence rate in rural areas is high. The clinical manifestations are paroxysmal abdominal pain and vomiting around the umbilicus, which may have a history of aphids or spit mites.

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