left ventricular chamber volume increase
Introduction
Introduction The left ventricular cavity volume increased, the posterior anterior position, the left ventricular segment extended, and the apex extended to the left. The apex of the apex is displayed in the stomach cavity, and becomes dull and left shifting, and the midline of the clavicle. The normal apex is at the level of the iliac crest and within the midline of the clavicle. The left ventricle segment is round and the heart is depressed. As the left ventricular segment is extended, the opposite beat point is moved up. When the increase is significant, the right ventricle can be pushed to move the lower right part of the right edge of the heart to the right and bulge, or the left atrium can be pushed to shift backwards and upwards. At the same time, the heart can be rotated to the right, making the depression of the heart more obvious.
Cause
Cause
Mainly caused by coronary artery obstruction, myocardial infarction, myocardial fibrosis and left ventricular ventricular aneurysm.
Examine
an examination
Related inspection
Electrocardiogram
The myocardial contractility of the lesion area is lost, and reverse pulsation can occur. The common clinical manifestations are shortness of breath, left heart failure, angina pectoris, arrhythmia and systemic arterial embolism. The severity of clinical symptoms is closely related to the number and functional status of the myocardium in the normal part of the left ventricle.
Diagnosis
Differential diagnosis
Differential diagnosis of left ventricular volume increase: left ventricular aneurysm: about 85% in the anterior lateral approach to the apical region, a small number of cases can be located in the heart. The ventricular wall of the lesion area became thinner, showing a white fibrous scar with a clear boundary, and the local epicardium and the pericardium were tightly adhered. About half of the cases have a wall thrombus on the endocardial surface, sometimes showing calcification. Most of the coronary artery obstruction lesions are confined to the left anterior descending artery, but several blood vessels may also be involved. The left ventricular cavity volume increases, and the normal part of the heart muscle is thick. Myocardial infarction: refers to ischemic necrosis of the myocardium. On the basis of coronary artery disease, the blood flow of the coronary artery is drastically reduced or interrupted, causing severe and persistent acute ischemia of the corresponding myocardium, eventually leading to myocardial ischemia. Necrosis.
Myocardial fibrosis: a result of myocardial ischemia and hypoxia caused by moderate to severe coronary atherosclerotic stenosis caused by myocardial fiber persistence and/or recurrence, leading to progressive development of heart failure IHD, a chronic ischemic heart disease. The myocardial contractility of the lesion area is lost, and reverse pulsation can occur. The common clinical manifestations are shortness of breath, left heart failure, angina pectoris, arrhythmia and systemic arterial embolism. The severity of clinical symptoms is closely related to the number and functional status of the myocardium in the normal part of the left ventricle.
