premature pubic hair development
Introduction
Introduction Early development of pubic hair: Under normal circumstances, girls develop pubic hair several months after breast development; boys develop pubic hair only months after penis and testicular development. Early developmental pubic hair can occur at any age, with more girls than boys. The amount of pubic hair is small, sometimes there are manes, but other sexual characteristics develop normally. Most foods are consumed by children's health foods that contain hormones.
Cause
Cause
The cause of early development of pubic hair:
Excessive light is one of the important reasons for inducing precocious puberty in children, because light affects the normal work of the endocrine organ pineal gland in the brain. One of the functions of the pineal gland is to secrete a large amount of melatonin when the human body enters a sleep state at night. This hormone is secreted most vigorously from 11 o'clock in the middle of the night to the next morning, and the light source stops secreting after dawn. The pineal gland has a characteristic that as soon as the eyeball sees the light source, melatonin is inhibited or stopped. If children are exposed to excessive light, it will reduce the secretion of melatonin in the pineal gland, which may lead to early secretion of follicle stimulating hormone, which leads to precocious puberty in children. The prevalence of precocious puberty in children is increasing year by year, but the actual premature puberty of children is less than 10%. 90% of children with precocious puberty are affected by external factors, and excessive light stimulation should be paid attention to, especially for children at night. When sleeping, if there is no special situation, it is best not to turn on the lights, and to ensure adequate sleep as much as possible. In addition, it is necessary to avoid the light stimulation of the computer display for a long time, so as to avoid the premature puberty of the child.
There are many causes of precocious puberty in children. The functions of the thalamus-pituitary-gonadal axis can be started in advance, and they can be divided into central (true) and peripheral (false).
Examine
an examination
Related inspection
Child growth and development test growth hormone
Examination and diagnosis of early development of pubic hair:
Detailed and complete medical history, including sexual development, vaginal bleeding, and endocrine drugs. Determination of follicle-stimulating hormone FSH and LH in blood by radioimmunoassay is helpful to distinguish between premature and precocious puberty. In children with precocious puberty, there may be an enlarged ovary and a cystic change. Suspected adrenal cortical disease, can be used for posterior wall inflation imaging. The positive lateral image of the skull, the size of the saddle is observed to exclude tumors.
Diagnosis
Differential diagnosis
Symptoms of early pubic hair that are confusing:
Mane and pubic hair shedding: Sheehan's syndrome - anterior pituitary hypofunction (Simon-Siehan syndrome) is a deficiency of pituitary hormone secretion caused by multiple causes, secondary hypogonadal, thyroid, adrenal insufficiency Clinical signs.
Adrenal insufficiency - When most of the adrenal glands on both sides are destroyed, there is a manifestation of various corticosteroid dysfunction, called adrenal insufficiency. Both the above-mentioned diseases can cause pubic hair and pubic hair loss.
No whisker hair and pubic hair: seen in Klinefelter syndrome. Also known as congenital testicular hypoplasia or Klinefelter syndrome. The typical karyotype is 47,XXY. The phenotype is characterized by testicular hypoplasia. The slender figure is caused by the increase in the distance from the heel to the phalanx.
Male mammary gland development, pubic hair is female-type distribution, penis and testicles are small. Severe cases are accompanied by mental retardation, cryptorchidism and hypospadias.
The vulva is naive and has no pubic hair: it is common in patients with Turner syndrome, also known as congenital ovarian hypoplasia syndrome, which is the only known sex chromosome disease. The patient's phenotype is female, short stature, normal intelligence, but often lower than his compatriots, the face is triangular, often with ptosis and internal suture, etc., the maxillary narrow, the lower jaw is small and retracted, the mouth is down and shark The mouth of the neck, the hairline of the neck is very low, can extend to the shoulders, about 50% of patients have a neck, that is, excess wing-like skin, shoulder width, chest width as a shield, poor development of nipples and breasts, two nipples The width and elbow valgus are very typical in this disease. The fourth and fifth metacarpals are short and inward, and often have nail dysplasia. Lymphatic swelling in the instep of infancy is very special.
Abnormalities in the genitourinary system are mainly poor ovarian development (somatogonous glands), no follicular formation, uterine hypoplasia, often due to primary amenorrhea. Patients with low ovarian function have few pubic hairs, no hair, and the external genitalia are naive. In addition, approximately 1/2 of patients have aortic stenosis and horseshoe kidney deformity.
Detailed and complete medical history, including sexual development, vaginal bleeding, and endocrine drugs. Determination of follicle-stimulating hormone FSH and LH in blood by radioimmunoassay is helpful to distinguish between premature and precocious puberty. In children with precocious puberty, there may be an enlarged ovary and a cystic change. Suspected adrenal cortical disease, can be used for posterior wall inflation imaging. The positive lateral image of the skull, the size of the saddle is observed to exclude tumors.
