Thinning of armpit hair
Introduction
Introduction The clinical manifestations of mane hair loss due to certain diseases. It is one of the clinical symptoms of facial unilateral atrophy. Progressive hemifacial atrophy (progressivehemifacialatrophy) is also called Parry-Romberg syndrome, a progressive unilateral tissue dystrophic disease, a small number of lesions involving the limbs or body, called progressive half atrophy Its clinical features are chronic progressive atrophic muscle fibers of focal subcutaneous fat and connective tissue on one side of the face and are not affected by severe cartilage and bone. Most scholars believe that this disease and sympathetic dysfunction are related to various causes of sympathetic nerve damage, causing facial tissue neurotrophic disorders, and finally lead to facial tissue atrophy. Other doctrines involve local or systemic infection damage, genetic degeneration of trigeminal connective tissue disease.
Cause
Cause
The cause of the scarring of the mane:
The cause of this disease is not yet clear. Symptoms of cervical sympathetic disorders, including Horner syndrome, are generally considered to be associated with central or peripheral damage to the autonomic nervous system.
In recent years, some patients have found that some patients often have dysplasia, congenital aneurysm, brain hypoplasia, etc. It is speculated that genetic factors cause abnormal embryogenesis, which may also be related to this disease. In addition, a small number of this disease can be secondary to certain infections such as polio, trauma, autocrine diseases with endocrine dysfunction.
Examine
an examination
Related inspection
Endocrine function test
Examination and diagnosis of scarring of the mane:
The diagnosis is based on the special facial morphology and imaging changes of the disease. When the patient has a typical unilateral atrophy, especially when the subcutaneous fat atrophy occasionally affects the head, neck, shoulders and limbs, the muscle strength is not affected.
1. Adolescents who are born before the age of 20 are occasionally seen in women who are diagnosed within 1 year of age. Insidious onset, slow progress, the process of atrophy can start at any part of the face, such as a side of the cheeks, etc., more common in the upper part and the upper part. The starting point is often strip-shaped and parallel to the midline; the skin is dry and wrinkled and the hair is detached. The lesion is called "knife mark". The lesion develops slowly to half of the face. The forehead of the eyelids, the cheeks of the ankle, the tongue and gums, etc. . Occasionally, it can affect the facial head, neck, shoulders, or other parts of the body to cause pain or sensory disturbances on the cheeks. A few onsets are seizures.
2. The ward is characterized by subcutaneous fat and connective tissue atrophy, skin atrophy and wrinkles, often accompanied by hair loss, hyperpigmentation, leukoplakia, telangiectasia, increased sweat secretion or reduced salivary secretion, sacral frontal bone, etc., and normal skin There is a clear dividing line.
3. Some cases show pupillary changes, iris pigmentation, eyeball retraction or protrusion, eyeball inflammation, secondary glaucoma facial pain or mild side sensation, facial muscle twitching, and endocrine disorders, which may progress with the course of the disease. Facial atrophy and focal lipoatrophy, often accompanied by skin hardening in a certain part of the body involving the limbs and trunk of the lesions, the limbs become thinner and shorter, the breasts become smaller, the bristles become thinner, the organs become smaller, but the muscles The force is normal. Some atrophy invades the contralateral limbs called cross-lateral atrophy.
Diagnosis
Differential diagnosis
Identification of symptoms that are less confusing when the hair is thinner:
1. No mane: The mane is located under the armpit and is the result of the adrenal gland beginning to secrete androgen. Once the armpits grow out of the armpits, they are often sweaty in the armpits. Mane is one of the first signs of your puberty. Everyone has a long hair, it helps to absorb sweat to prevent sweat from flowing down. No whisker hair and pubic hair were found in Klinefelter syndrome. Also known as congenital testicular hypoplasia or Klinefelter syndrome. The typical karyotype is 47,XXY. The phenotypic characteristics are testicular hypoplasia. The slender figure is caused by the increase in the distance from the heel to the phalanx. Male mammary gland development, pubic hair is female-type distribution, penis and testicles are small. Severe cases are accompanied by mental retardation, cryptorchidism and hypospadias.
2, mane and pubic hair shedding: Xi's syndrome - anterior pituitary hypofunction (Simon-Shan Han syndrome) is caused by a variety of causes of pituitary hormone secretion, secondary gonads, thyroid, adrenal insufficiency The clinical signs presented. Adrenal insufficiency - When most of the adrenal glands on both sides are destroyed, there is a manifestation of various corticosteroid dysfunction, called adrenal insufficiency. Both the above-mentioned diseases can cause pubic hair and pubic hair loss.
The diagnosis is based on the special facial morphology and imaging changes of the disease. When the patient has a typical unilateral atrophy, especially when the subcutaneous fat atrophy occasionally affects the head, neck, shoulders and limbs, the muscle strength is not affected.
1. Adolescents who are born before the age of 20 are occasionally seen in women who are diagnosed within 1 year of age. Insidious onset, slow progress, the process of atrophy can start at any part of the face, such as a side of the cheeks, etc., more common in the upper part and the upper part. The starting point is often strip-shaped and parallel to the midline; the skin is dry and wrinkled and the hair is detached. The lesion is called "knife mark". The lesion develops slowly to half of the face. The forehead of the eyelids, the cheeks of the ankle, the tongue and gums, etc. . Occasionally, it can affect the facial head, neck, shoulders, or other parts of the body to cause pain or sensory disturbances on the cheeks. A few onsets are seizures.
2. The ward is characterized by subcutaneous fat and connective tissue atrophy, skin atrophy and wrinkles, often accompanied by hair loss, hyperpigmentation, leukoplakia, telangiectasia, increased sweat secretion or reduced salivary secretion, sacral frontal bone, etc., and normal skin There is a clear dividing line.
3. Some cases show pupillary changes, iris pigmentation, eyeball retraction or protrusion, eyeball inflammation, secondary glaucoma facial pain or mild side sensation, facial muscle twitching, and endocrine disorders, which may progress with the course of the disease. Facial atrophy and focal lipoatrophy, often accompanied by skin hardening in a certain part of the body involving the limbs and trunk of the lesions, the limbs become thinner and shorter, the breasts become smaller, the bristles become thinner, the organs become smaller, but the muscles The force is normal. Some atrophy invades the contralateral limbs called cross-lateral atrophy.
