Scattered gray-white papules in the pharynx
Introduction
Introduction The presence of scattered grayish white herpes in the pharynx is one of the important clinical manifestations of enterovirus infection.
Cause
Cause
The cause of the grayish white herpes in the pharynx
The virus invades from the oropharynx and multiplies in local mucosa or lymphoid tissue, causing local symptoms. The virus then invades the local lymph nodes and thereby enters the blood circulation leading to viremia. The virus spreads with the bloodstream to the whole body organs such as the central nervous system, the skin and mucous membranes, the heart, the respiratory organs, the liver, the muscles, the meat, etc., and further propagates in these areas and causes lesions.
Pathological changes vary depending on the organ invaded. The main lesions are parenchymal degeneration and necrosis with inflammatory cell infiltration.
Examine
an examination
Related inspection
Otolaryngology CT examination
Examination of the pharyngeal herpes in the grayish white herpes
1. Virus isolation: The isolation of the virus from the patient's body fluid (pleural fluid, pericardial fluid, cerebrospinal fluid, blood, herpes fluid, etc.) or biopsy and autopsy tissue has diagnostic value, but the virus can not be diagnosed from the throat swab or feces alone. If the same type of virus is repeatedly isolated from the throat swab or feces of the patients with the above clinical symptoms, and the same virus is detected from the surrounding patients with the same disease, and the virus isolation rate is much higher than the normal population, there is a diagnosis. Reference value.
2. Serological examination: the serum neutralizing antibody titer increased more than 4 times in the early and recovery period, and has diagnostic value.
Diagnosis
Differential diagnosis
There are symptoms in the pharynx that are scattered in the grayish white herpes.
Oropharynx vesicular lesions: Oropharynx vesicular lesions are a clinical condition caused by B herpesvirus infection.
B virus disease (B virus disease) is also known as herpes virus simiae, which is named after the infection of monkeys. Skin damage, lymphadenitis and flu-like syndrome can occur in people who are infected with BV monkeys or scratches, and even toxic encephalitis.
Foreign body sensation in the pharynx: Foreign body sensation in the pharynx is not a unique disease, but a combination of some symptoms. It includes a feeling of blockage, pressure, leafiness, stenosis, dryness, burning, itching, anthillness or other discomfort. Its causes are numerous, sometimes quite complex, organic and functional. Functionality is also known as pharyngeal paraesthesia, laryngeal paraesthesia, plum nuclear gas, pharyngeal neurosis, and sputum.
Pharyngeal ulcer: ulcer is a localized defect or ulceration of the skin or mucosal surface tissue. The surface is often covered with pus, necrotic tissue or ecdysis. After the scar, there is scar, which can be caused by infection, trauma, nodules or tumor. Due to the same, its size, shape, depth, development process, etc. are also inconsistent. Often combined with chronic infection, may not heal for a long time. There are many diseases that cause pharyngeal ulcers, and the reasons are different. In addition to some specific infections (diphtheria, tuberculosis, syphilis, induration, etc.) and pharyngeal tumors, the more common ulcer pseudomembranous lesions.
1. Virus isolation: The isolation of the virus from the patient's body fluid (pleural fluid, pericardial fluid, cerebrospinal fluid, blood, herpes fluid, etc.) or biopsy and autopsy tissue has diagnostic value, but the virus can not be diagnosed from the throat swab or feces alone. If the same type of virus is repeatedly isolated from the throat swab or feces of the patients with the above clinical symptoms, and the same virus is detected from the surrounding patients with the same disease, and the virus isolation rate is much higher than the normal population, there is a diagnosis. Reference value.
2. Serological examination: the serum neutralizing antibody titer increased more than 4 times in the early and recovery period, and has diagnostic value.
