Calf Ulcer
Introduction
Introduction Calf ulcers, also known as chronic calf ulcers, varicose ulcers, and stagnation ulcers, are common skin diseases. The disease is due to varicose veins, increased venous pressure, capillary damage, local circulatory disorders, small arteries and lymphatic obstruction, resulting in local tissue malnutrition, reduced oxygenation, or traumatic infection to promote ulcer formation. It belongs to the category of Chinese medicine "hemorrhoids", "trousers sores" and "skirt sores". Calf ulcers are generally more common in patients over 40 years old, mostly due to long standing or weight loss, resulting in vascular dysfunction, local qi and blood, affecting the return of veins in the lower extremities; or due to skin damage in the lower extremities, and eczema , causing hot and humid bets, qi and blood stagnation of the meridians.
Cause
Cause
Due to varicose veins or venous insufficiency, local blood stasis, followed by shallow ulcers, size and shape are uncertain, gradually enlarge and deepen. The surrounding skin is pigmented and sees scales and eczema-like changes. The sore surface is dark red, skunk, and unhealed. Conscious pain, sometimes redness and pus. Lower limbs with acidosis or with fever.
The disease is due to increased pulse pressure, capillary damage, local circulatory disorders, small arteries and lymphatic obstruction, resulting in local tissue malnutrition, reduced oxygenation, or traumatic infection to promote ulcer formation. Belong to the Chinese medicine "hemorrhoids", "trousers sores", "skirt sores" and other diseases
Examine
an examination
Related inspection
Blood routine spore agglutination test
Calf ulcers are generally more common in patients over 40 years old, mostly due to long standing or weight loss, resulting in vascular dysfunction, local qi and blood, affecting the return of veins in the lower extremities; or due to skin damage in the lower extremities, and eczema , causing hot and humid bets, qi and blood stagnation of the meridians.
Occurs in the inside of the lower third of the lower leg. At first, due to varicose veins or venous insufficiency, local blood stasis, followed by shallow ulcers, size and shape are uncertain, gradually deepening and deepening. The surrounding skin is pigmented and sees scales and eczema-like changes. The sore surface is dark red, skunk, and unhealed. Conscious pain, sometimes redness and pus. Lower limbs with acidosis or with fever.
Diagnosis
Differential diagnosis
Differential diagnosis:
1. Renault sign: more common in women, occurs in the hands caused by paroxysmal small artery sputum caused by the skin of both ends of the skin is pale, that is, purplish redness and swelling, conscious numbness or tingling, repeated attacks on the extremities can occur Necrotic ulcer.
2. Varicose ulcers: It can be seen in adults who often stand or use too much force in the lower extremities. The ulcers occur after varicose veins of the lower extremities. The skin of the affected limbs is eczema-like, and chronic ulcers often form near the deep joints. Chronic pigmentation is often common around.
3. Allergic vasculitis: often pleomorphic damage can occur skin ulcers.
4. Occlusive thrombotic vasculitis: occurs in the lower extremity lower extremity numbness pale painful intermittent pain, broken foot dorsal artery pulsation weakened or disappeared, severe cases of acral ulcer and necrosis as dry gangrene. This disease is common among young men who smoke.
5. Wegener granulomatosis: an immune response to necrotizing vasculitis, may occur in the pathogenesis of adults, more men than women, systemic symptoms of the nasopharyngeal trachea occurred several nodules nasal nodules. Usually ulceration can form a ulcer that can invade the pulmonary parenchymal granuloma, which can occur in the nasal cavity and the tongue, and can occur in ulcers or over-perforated skin, showing a group of nodules. Most cases of necrotizing ulcers can occur in the center of the extremities, and there may be erythematous epidemic hemorrhagic rash. Focal necrotizing nephritis is a serious symptom of this disease. Many patients die of renal failure. Eosinophils in the blood can be Up to 0.80. The above pathology shows changes in arterial venous necrotic vasculitis and necrotizing granuloma.
6. Fatal midline granuloma: This disease occurs mostly in men aged 20-50 years. The cause is unknown. The damage occurs on the face, especially the progressive destructive granulomatous ulcer in the nose. It can develop on the nasal septum and face and cranium. The bottom can be destroyed by destruction.
7. gangrenous pyoderma: the etiology of this disease is unknown, often complicated with other diseases, for the recurrence of degenerative skin ulcers, pustular blister, small nodules, central necrosis, ulcers and ulcers, clear skin, purple skin Red edema and tissue down, sneak damage and partial pus healing and new rash appeared.
