indigestion
Introduction
Introduction Indigestion is a disease caused by gastric dysmotility, and it also includes gastroparesis and esophageal reflux disease with poor gastric motility. Indigestion is mainly divided into functional dyspepsia and organic dyspepsia. It belongs to the category of "sputum", "stomach pain" and "noisy" of traditional Chinese medicine. Its disease is in the stomach, involving organs such as liver and spleen. It should be treated according to syndrome differentiation, spleen and stomach, and liver and qi.
Cause
Cause
Occasional indigestion can be caused by eating too much, drinking too much, and taking painkillers such as aspirin. Eating when you are nervous or eating a diet that you are not used to can also cause. Chronic persistent dyspepsia can be neurological, that is, caused by mental factors, or can be caused by obsessive diseases such as chronic gastritis, stomach and duodenal ulcer, chronic hepatitis, and after gallbladder removal. Patients can also often suffer from indigestion. Either way, because the stomach lacks motivation, it can't work normally, and the food stays in the stomach for too long.
Examine
an examination
Related inspection
Gastric sputum meal urinary bile acid general examination of fecal plant cells and plant fibers McNealy-McCallister test
Loss of appetite, abdominal fullness after eating, abdominal pressure and/or abdominal pain, can be radiated to the chest, suffocating, heartburn, mild nausea, vomiting, thick and greasy tongue coating.
Pain or discomfort in the upper abdomen may be accompanied by an increase in hernia and bowel sounds. Some patients eat more to make the pain worse, while others have the opposite, eating to reduce the pain. Loss of appetite, nausea, constipation, bloating and flatulence.
Diagnosis
Differential diagnosis
(1) ulcer-like dyspepsia, which is characterized by the symptoms of peptic ulcer without the presence of ulcers. Recent studies have found that patients who are often exposed to stress may develop stress reactions with intermittent increases in gastric acid excretion. In addition, dysmotility increases the effect of gastric acid on mucosal damage. Therefore, this type of patient can be improved by eating or taking an H-receptor antagonist.
(2) dysmotility-like dyspepsia, which is characterized by clinical manifestations of gastric retention symptoms. Patients have difficulty in positioning upper abdominal pain or discomfort, often caused by eating or aggravation after meals, and there is a post-prandial upper abdomen Bulging, early satiety, nausea or vomiting, poor appetite, etc.
(3) Patients with specific dyspepsia, who have FD symptoms but do not meet the above two groups of characteristic dyspepsia.
Organic dyspepsia: After examination, it can be clearly identified that the symptoms of dyspepsia caused by an organ lesion, such as liver disease, biliary tract disease, pancreatic disease, diabetes and so on. For these patients, the treatment is mainly for the treatment of the cause, supplementing the digestive enzymes or improving the gastric motility to relieve the symptoms of dyspepsia. Loss of appetite, abdominal fullness after eating, abdominal pressure and/or abdominal pain, can be radiated to the chest, suffocating, heartburn, mild nausea, vomiting, thick and greasy tongue coating.
