bacterial endocarditis
Introduction
Introduction Bacterial infections of bacilli, cocci, Gram-positive and negative bacteria, various organs of the body, bacterial infections can occur in all systems. Acute bacterial endocarditis, 50-60% occurs in normal heart valves, valves and tendons can have ulcer perforation, chordae rupture and large and brittle mites, sputum exfoliation into bacterial emboli, leading to embolism and migration Sexual abscess.
Cause
Cause
Cause: In most cases, the cause is from a bacterial infection. In acute cases, the cord-like tendon or valve matrix is rapidly destroyed and necrotic, leading to valve dysfunction and heart failure. Earthworms contain fibrous layers, platelets, red blood cells and white blood cells and bacteria. Often caused by bacteria with strong pathogenicity, Staphylococcus aureus, hemolytic streptococcus, pneumococcus, influenza bacillus, Proteus and Escherichia coli are common. These bacteria have strong virulence, acute onset, serious illness, and infections often occurring in other parts, which are part of systemic infections, such as meningitis, pneumonia, thrombophlebitis, etc., sometimes accompanied by metastatic suppuration of other organs. Lesion. Usually occurs in normal heart.
Examine
an examination
Related inspection
Cardiovascular electrocardiogram, blood routine, Doppler echocardiography, anti-double-stranded DNA antibody
1. The medical history is usually ambiguous and non-specific, more like systemic infection or inflammation than heart disease. Clinical symptoms depend on the organ involved and may reflect altered valve function. Sepsis and bacteremia; local infections of vascular embolism in joints, kidneys, muscles, heart muscle, lungs, and central nervous tissues; secondary immune-mediated diseases that affect joints and glomeruli.
2. Heart murmur may occur at the end of the disease. The heart murmur may be a heart sound or a heart sound, and the intensity changes at any time. Vascular embolization can cause myocardial infarction or myocarditis and lead to arrhythmia. Some cases have symptoms of left heart failure.
3, in addition to myocardial symptoms, but also include narcolepsy, anorexia, weightlessness, intermittent heat, lameness (often changed with the variety), muscle tension, ecchymosis or congestive bleeding, dyspnea and seizures. Direct spread of infection to one or more joints can lead to purulent polyarthritis, or persistent antigenic stimulation causes deposition of immune complexes in the joint membrane and non-purulent (non-necrotic) immune-mediated polyarthritis .
Diagnosis
Differential diagnosis
Differential diagnosis of bacterial endocarditis:
1. Subacute bacterial endocarditis: often caused by bacterial infection based on the original heart disease (such as heart valve disease congenital heart disease). The pathogens are mostly Staphylococcus aureus, and a few are Escherichia coli. It belongs to the category of "warm disease" of Chinese medicine.
2. Acute bacterial endocarditis: 50-60% occurs in normal heart valves, valves and tendons may have ulcer perforation, chordae rupture and large and brittle mites, sputum exfoliation as bacterial emboli, leading to embolism And a migratory abscess.
an examination
1. The medical history is usually ambiguous and non-specific, more like systemic infection or inflammation than heart disease. Clinical symptoms depend on the organ involved and may reflect altered valve function. Sepsis and bacteremia; local infections of vascular embolism in joints, kidneys, muscles, heart muscle, lungs, and central nervous tissues; secondary immune-mediated diseases that affect joints and glomeruli.
2. Heart murmur may occur at the end of the disease. The heart murmur may be a heart sound or a heart sound, and the intensity changes at any time. Vascular embolization can cause myocardial infarction or myocarditis and lead to arrhythmia. Some cases have symptoms of left heart failure.
3, in addition to myocardial symptoms, but also include narcolepsy, anorexia, weightlessness, intermittent heat, lameness (often changed with the variety), muscle tension, ecchymosis or congestive bleeding, dyspnea and seizures. Direct spread of infection to one or more joints can lead to purulent polyarthritis, or persistent antigenic stimulation causes deposition of immune complexes in the joint membrane and non-purulent (non-necrotic) immune-mediated polyarthritis .
