Myocardium pale and relaxed
Introduction
Introduction Myocardial gray and relaxation is a type of myocardial damage, which may be related to immune myocardial damage after viral infection, and is generally seen in ultrasonography of dilated cardiomyopathy. Can be used as a differential diagnosis with other cardiomyopathy.
Cause
Cause
The etiology of this disease has not been known so far, and it is believed to be caused by immune myocardial damage occurring after viral infection, and is the main theory of dilated cardiomyopathy.
Examine
an examination
Related inspection
Cardiovascular angiography Doppler echocardiogram electrocardiogram
Ultrasound examination of dilated cardiomyopathy can be found: the weight of the heart is increased, the heart chamber is enlarged, the myocardium is gray and relaxed; the thickness of the wall is almost normal, the endocardium can also be thickened, and there may be a wall thrombus in the heart chamber, often with myocardial fibrosis. It can also damage the heart wall and the heart pacing system can also be invaded. Dilated cardiomyopathy can occur at all ages, but it is mostly in middle age. The onset was slow, and the heart was enlarged at the initial examination, and the heart function was compensated without conscious discomfort. Symptoms gradually appear after a period of time, sometimes more than 10 years. Symptoms are mainly congestive heart failure, with air urgency and edema being the most common.
Further detailed medical history and physical examination, echocardiography, X-ray examination, electrocardiogram, myocardial enzymes and other examinations are helpful for diagnosis and classification, and the diagnosis is sometimes dependent on myocardial biopsy and cardiovascular angiography.
Diagnosis
Differential diagnosis
Myocardial infarction: refers to the interruption of coronary blood flow on the basis of coronary artery disease, causing severe and long-lasting acute ischemia in the corresponding myocardium, eventually leading to myocardial ischemic necrosis.
Myocardial hypoxia: The heart is deprived of oxygen due to insufficient blood supply. The main manifestations are: palpitations, heart discomfort, sometimes heart pain or appreciation of colic; shortness of breath, exercise, fullness or agitation is more serious, the body is weak; in severe cases, short-term shock.
Myocardial abscess: Myocardial edema is one of the main clinical symptoms of dilated cardiomyopathy.
Myocardial stunning, also known as myocardial dysfunction after ischemia, means that myocardial transient ischemia has not caused myocardial necrosis, but mechanical resilience after reperfusion returns to normal blood flow requires hours, days or weeks. The phenomenon of complete recovery.
Cardiac hypertrophy: This is a slower but more effective compensatory function, mainly in the case of long-term stress overload, increased myocardial volume, increased contractility, allowing the heart to maintain a normal blood circulation, and at the same time Reserve power. However, this compensatory function also has its disadvantages, mainly because of hypertrophic myocardial aerobic increase, and the coronary blood supply is often unable to be met, resulting in myocardial ischemia, which will eventually lead to a decline in myocardial contractility. Hypertrophic cardiomyopathy is characterized by cardiac hypertrophy. It is characterized by ventricular muscle hypertrophy, typically in the left ventricle, with interventricular septum, occasionally concentric hypertrophy.
Ultrasound examination of dilated cardiomyopathy can be found: the weight of the heart is increased, the heart chamber is enlarged, the myocardium is gray and relaxed; the thickness of the wall is almost normal, the endocardium can also be thickened, and there may be a wall thrombus in the heart chamber, often with myocardial fibrosis. It can also damage the heart wall and the heart pacing system can also be invaded.
Dilated cardiomyopathy can occur at all ages, but it is mostly in middle age. The onset was slow, and the heart was enlarged at the initial examination, and the heart function was compensated without conscious discomfort. Symptoms gradually appear after a period of time, sometimes more than 10 years. Symptoms are mainly congestive heart failure, with air urgency and edema being the most common. Further detailed medical history and physical examination, echocardiography, X-ray examination, electrocardiogram, myocardial enzymes and other examinations are helpful for diagnosis and classification, and the diagnosis is sometimes dependent on myocardial biopsy and cardiovascular angiography.
