Increased urobilinogen excretion
Introduction
Introduction Increased urinary tract excretion is a symptom of shunting hyperbilirubinemia syndrome. The shunt hyperbilirubinemia syndrome is Israel syndrome. Excessive bilirubin produced by bone marrow red blood cells or precursors, or directly caused by destruction and production of erythroline or tetrapyrrole precursors. Excessive bilirubin is produced by bone marrow red blood cells or precursors, or directly by the destruction and production of ferrene or tetrapyrrole precursors. Pay attention to proper rest, do not master the combination of movement and rest, rest well, is conducive to the recovery of fatigue; exercise can enhance physical strength, enhance disease resistance, the combination of the two, can better recover.
Cause
Cause
Excessive bilirubin is produced by bone marrow red blood cells or precursors, or directly by the destruction and production of ferrene or tetrapyrrole precursors. When red blood cells in the blood die, the hemoglobin (Heme) of hemoglobin in the red blood cells is collected in the liver's Kupffer cells and the spleen is converted into bilirubin. After treatment with the liver, bilirubin is secreted into the duodenum with bile, and finally passes through the digestive system and is excreted together with the feces.
Examine
an examination
Related inspection
Urine routine renal function test urinary bile test urinary triad examination
In the presence of jaundice, serum total bilirubin and direct bilirubin should be examined to distinguish the type of elevated bilirubin, as well as urinary bilirubin, urobilinogen and liver function.
Auxiliary examination: blood routine, urine routine, jaundice index, serum bilirubin quantitative test; urine bilirubin, urobilinogen, urinary bilirubin examination; serum enzymology; blood cholesterol and cholesterol ester determination; immunological examination X-ray examination; B-mode ultrasound examination; radionuclide examination; liver biopsy; laparoscopy.
Diagnosis
Differential diagnosis
It should be differentiated from the increase of urobilinogen: urinary biliary is derived from the combination of bilirubin. In combination with bilirubin in the lower part of the small intestine and in the colon, it is decoupled by the action of intestinal bacteria. After several stages of reduction, bilirubin becomes urinary biliary and then excreted with feces. A part of urinary biliary is absorbed into the portal vein from the intestine, most of which is taken up by the liver cells and then discharged into the intestinal fluid (intestinal hepatic circulation), and part of it enters the systemic circulation from the portal vein and is excreted from the urine through the kidney. A variety of factors can cause an increase in urinary biliary.
