gastric mucosal congestion
Introduction
Introduction Gastroscopic examination of acute gastritis revealed extensive hyperemia of the mucosa, edema, erosion, hemorrhage, sometimes visible mucus spots on the mucosal surface or bile reflux. When Hp is infected with gastritis, micronodular formation of gastric mucosa can also be seen. Allergic reactions caused by mood swings, stress states and various factors in the body can cause disease as endogenous stimuli. Acute gastritis (acute gestritis) is an acute inflammation of the gastric mucosa caused by no cause. Clinically, it can be divided into simple, erosive, corrosive and suppurative, and simplest is the most common. Some people divide it into acute external and acute endogenous. Gastric mucosal congestion and edema is caused by reflux of bile, pancreatic juice and intestinal alkaline fluid into the stomach. It is more common in right heart failure caused by various cardiovascular diseases and portal hypertension caused by various causes.
Cause
Cause
1 Physical factors: too cold, overheated foods and beverages, strong tea, coffee, spirits, irritating condiments, too rough foods, drugs (especially non-steroidal anti-inflammatory drugs such as aspirin, indomethacin, etc.) Both can stimulate the gastric mucosa and destroy the mucosal barrier.
2 Chemical factors: Aspirin and other drugs can also interfere with the synthesis of threose protein in gastric mucosal epithelial cells, so that the mucus in the stomach is reduced, the protective effect of lipoprotein membrane is weakened, and the hydrogen ions in the gastric cavity are reversely diffused, resulting in the release of mucosal lamina propria. Histamine, increased vascular permeability, resulting in gastric mucosal congestion, edema, erosion and hemorrhage and other pathological processes, prostaglandin synthesis is inhibited, and gastric mucosal repair is also affected.
3 Biological factors: bacteria and their toxins. Common pathogens are Salmonella, halophilic bacteria, pathogenic Escherichia coli, etc. Common toxins are Staphylococcus aureus or toxin bacilli, especially in the former. Gastroenteritis or a combination of enteritis and acute gastroenteritis can occur after eating food contaminated with bacteria or toxins for several hours. Staphylococcal and its toxins are combined with enteritis and this is acute gastroenteritis. The onset of staphylococcal and its toxins is faster. In recent years, there have been a few cases of this disease caused by viral infection.
4 Mental and neurological factors: mental and neurological disorders, critical conditions of various acute and severe diseases, and abnormal (allergic) reactions of the body can cause acute inflammatory damage to the gastric mucosa.
5 Intragastric foreign body or stomach stone, stomach area radiation therapy can be used as an exogenous stimulus, leading to this disease. Allergic reactions caused by mood swings, stress states and various factors in the body can cause disease as endogenous stimuli.
Examine
an examination
Related inspection
Gastric sputum meal angiography fiber gastroscopy gastric microscopy gas sputum double contrast angiography gastric mucosa biopsy
Gastroscopy: For the most valuable, safe and reliable diagnostic methods, the gastric mucosal lesions and their extent can be directly observed.
X-ray barium meal imaging: Most gastritis lesions in the mucosal surface, barium meal imaging is difficult to have a positive finding.
Stool blackening or fecal occult blood test positive: indicating bleeding in the gastric mucosa.
Diagnosis
Differential diagnosis
Gastric mucosal damage: The entire inner surface of the stomach is covered with a layer of mucosal tissue that secretes mucus, forming a mucous-mucosal barrier of the stomach to protect the stomach tissue. Gastric mucosal injury is more common in gastritis and gastric ulcer.
Atrophic changes in gastric mucosa: atrophic changes in gastric mucosa are clinical manifestations of gastric mucosal inflammatory lesions and chronic atrophic gastritis.
Thickening of gastric mucosa: one of the clinical manifestations of "chronic atrophic gastritis" (intestinal, atypical hyperplasia), chronic atrophic gastritis is a common stomach disease. Arteriosclerosis, insufficient blood flow to the stomach, and hobby of tobacco and tea are all likely to impair the barrier function of the gastric mucosa and cause chronic atrophic gastritis. In atrophic gastritis, the gastric mucosa is atrophied and replaced by intestinal epithelial cells, ie intestinal metaplasia; inflammation continues to evolve, cell growth is atypical, ie, metastasis; even cell proliferation and carcinogenesis.
Gastric mucosa shedding: The gastric mucosa is relatively relaxed. When the stomach contracts, the gastric mucosa near the pylorus is removed into the duodenal bulb. The clinical symptoms are symptoms of gastritis.
