stomatitis
Introduction
Introduction Stomatitis is inflammation of the oral mucosa, which can affect the buccal mucosa, tongue, gums, upper jaw and so on. It is more common in children, especially infants and young children. It can be caused by simple diseases or secondary diseases such as diarrhea, malnutrition, and chronic illness. Western medicine believes that bacterial infectious stomatitis often uses streptococcus and staphylococcus as the main pathogens. These bacteria in the case of acute infection, long-term diarrhea and other low body resistance, if the mouth is not clean, the bacteria cause massive proliferation, resulting in acute oral mucosal damage.
Cause
Cause
Western medicine believes that bacterial infectious stomatitis often uses streptococcus and staphylococcus as the main pathogens. These bacteria in the case of acute infection, long-term diarrhea and other low body resistance, if the mouth is not clean, the bacteria cause massive proliferation, resulting in acute oral mucosal damage. At the beginning, the oral mucosa is congested and edematous, followed by erosions or ulcers of varying sizes, with a grayish white pseudomembrane formed by thicker fibrous inflammatory exudates. The causative agent of herpetic stomatitis is herpes simplex virus. A local rash appears in the skin and rapidly oozes out to form blisters.
Examine
an examination
Related inspection
Oral X-ray examination
Blood routine examination
Bacterial stomatitis increased the total number of white blood cells, and the number of white blood cells in herpetic stomatitis was normal.
2. Histopathological examination
The tissue at the bottom of the vesicle was stained, and multinucleated giant cells were observed. Eosinophil particles were found in the nucleus. Under the electron microscope, the herpes simplex virus was found to be second only to the center of the nucleus.
Diagnosis
Differential diagnosis
The disease should be differentiated from herpes angina caused by Coxsackie A virus.
Diagnosis can be made based on symptoms and characteristic oral lesions: preferably by isolation of the virus from the lesion or by an increase in characteristic antibody titers, but these tests are not recommended for routine use. Differential diagnosis includes herpetic stomatitis (characterized by any season, presenting larger, longer lasting ulcers), recurrent aphthous ulcers and Bednar's mouth sores (rarely occurring in the pharynx, but generally without systemic symptoms). Coxsackie A group 10 virus also caused similar lesions (lymph node pharyngitis), but the oropharynx lesions were prominent, showing a white to yellow nodule, and the treatment was symptomatic.
