Fascia pain
Introduction
Introduction Whether the myogenic or fascia fascia soft tissue is injured by static or external force, or due to the influence of the surrounding environment, temperature and cold and dampness, the connective tissue lesions adhere to it, resulting in aseptic inflammation, resulting in ischemia and hypoxia. It directly affects the pain at the end of the nerve. Once the pain and spasm form a vicious circle, aseptic inflammation is further aggravated, resulting in increased pain in the local and surrounding tissues.
Cause
Cause
The fascia and other soft tissues are injured and cause disease. The main reason is that the muscle tissue is overloaded during exercise, causing different degrees of strain on the fascia, and symptoms of pain, soreness, numbness and swelling. The strained muscle tissue can improve the blood supply and oxygen supply by promoting the contraction of the antagonistic muscle, promoting the metabolites, allowing the excretion to relax and release, and achieving self-repairing purposes, but the fascia and spastic tissue do not have such self-repair. Features. Therefore, the fascia damage caused by excessive movement and static muscles must be artificially released or added to the biological oxidation mechanism to restore healing. Cold and humid, it is also an important external cause of myofascial pain. The temperature of the body surface drops, the cold and wet through the skin layer, the shallow fascia is tightened, the longer the attack time, the lower the temperature, the higher the humidity, the larger the range, resulting in poor circulation, insufficient blood supply and oxygen, and blocked metabolism, resulting in The chemical environment around the myofascial fascia changes, and the concentration of inflammatory chemical factors increases, causing lesions in the myofascial fascia.
Examine
an examination
Related inspection
CT examination of bone and joint and soft tissue MRI examination of whole body soft tissue
Muscle fascia soft tissue injury, secondary to periosteal and fibrotic inflammation, triggering part of bone hyperplasia, leukocyte infiltration, resulting in aseptic inflammation, causing local connective tissue lesions adhesion, resulting in ischemia and hypoxia, directly affecting To the nerve endings, it produces pain. Pain causes muscle-protective paralysis, and once the pain develops a vicious circle, aseptic inflammation is further aggravated, leading to an increase in pain in the surrounding tissue.
Diagnosis
Differential diagnosis
1. Necrotizing fasciitis: Necrotizing fasciitis is a rare and serious soft tissue infection. It is different from streptococcal necrosis and is often a mixed infection of various bacteria. Rea and Wyrick confirmed that pathogens include Gram-positive hemolytic streptococcus, Staphylococcus aureus, Gram-negative bacteria and anaerobic bacteria.
2, shallow fasciitis: the superficial fascia of the chest and lateral area and the neck, abdomen and upper limbs shallow fascia, containing fat, superficial blood vessels, lymphatic vessels, cutaneous nerves and breast. The individual thickness varies greatly, the front of the sternum is thinner, and the rest is thicker. Myofasciitis, also known as fibroinitis, can only be regarded as a comprehensive concept, which has rarely been adopted in recent years. Fibrositis is a condition in which some patients with low back pain have small nodules on the surface of the iliac spine or at the attachment of the diaphragm, accompanied by pain and tenderness, and sometimes found on the buttocks.
3, subcutaneous deep fascia progressive necrotic infection: surgical wound infection and other cellulitis often lead to bacteremia, the most serious is necrotizing fasciitis, for subcutaneous deep fascia and fat progressive necrotic infection, the process Infections often start from trauma (inconspicuous trauma) or localized redness, heat and pain in the surgery quickly spread outward. The color of the lesion changed from red to purple at 24 to 48 hours, and then turned blue to form blister and bullae containing yellow liquid. On the 4th to 5th day of the disease, the purple area began to be necrotic, and the skin with clear and necrotic borders fell off at 7 to 10 days, revealing extensive necrotic tissue under the skin. Patients with high fever and slow response are prone to bacteremia and sepsis. In fact, patients with TSLS are often accompanied by severe soft tissue infection.
