chordae rupture
Introduction
Introduction One end of the mitral valve is attached to the edge of the leaflet and the ventricular surface, and the other end is attached to the top of the papillary muscle and a few are directly connected to the posterior wall muscle of the left ventricle. The sacral cord is divided into 3 groups: one group is distributed on the edge of the leaf, which is thinner and more numerous. It mainly prevents the valve from turning to the left atrium when the left ventricle contracts. One group is attached to the anterior leaflet ventricular surface, which is thicker and less. Also known as the main chordae, it usually starts from the apex of the anterior lateral and posterior medial papillary muscles, ending at 4 to 5 points posterior to the anterior leaflet and 7 to 8 points at the anterior lateral, without the chordae in the posterior valve leaflet; 2 junctions.
Cause
Cause
The common cause of mitral chordae rupture is mitral valve prolapse, mucoid degeneration of the mitral valve, partial bulging between the prolapsed mitral leaf and the chord, and the length of the leaflet increases. Thin, long, twisted, fibrotic and thickened, due to abnormal chordae, mitral valve stress is uneven, chordae tendon rupture can occur when the chordae tendon tension increases.
Examine
an examination
Related inspection
Doppler echocardiography dynamic electrocardiogram (Holter monitoring)
Sudden dyspnea or aggravation of previous symptoms is a major feature of patients with chordae rupture. Almost all patients have full systolic murmurs in the apex of the heart, and some cases can smell the seagulls. Echocardiography is the most reliable method for diagnosing mitral chordae rupture; it is characterized by the mitral valve leaf motion of the diseased mitral valve, which breaks into the left atrium during the systolic phase and cannot be combined with another leaflet.
Diagnosis
Differential diagnosis
Mitral valve prolapse: The so-called mitral valve prolapse is a congenital problem, because the mitral valve is longer and longer, so when the ventricle contracts, because the pressure difference between the left atrium and the ventricle is too large, the valve is toward The direction of the atrium moves. Generally, about 10-20% of people have this phenomenon, but most people are asymptomatic. Only some people will have heart palpitations, chest tightness, poor breathing, and sometimes chest pain. In the situation, even someone will have a fainting situation.
