spinal bone changes
Introduction
Introduction Made up of bone tissue, dense and loose. Spinal bone changes refer to changes in the bones of the spine, and these changes are usually pathological changes. Spinal bone changes include spinal osteoporosis and spinal bone hyperplasia.
Cause
Cause
Osteoporosis: 1. Endocrine factors In women, osteoporosis is caused by estrogen deficiency, and men are caused by decreased testosterone levels due to decreased sexual function. 2. Genetic factors Osteoporosis is more common in whites, especially in Nordic countries, followed by Asians, while blacks are rare. Bone mineral density is an important indicator for the diagnosis of osteoporosis. The bone mineral density is mainly determined by genetic factors, and secondly by environmental factors.
Bone hyperplasia: early cartilage surface fragmentation, chondrocyte proliferation, cartilage surface longitudinal splitting, crystal deposition, cartilage repair, osteophyte hyperplasia; late cartilage destruction, manifested as cartilage sclerosis, cartilage disappearance and subchondral Focal osteonecrosis.
Examine
an examination
Related inspection
Spinal MRI examination of human leukocyte antigen B27 (HLA-B27) spinal exercise test
There is a strong feeling, limited activity, and the sound of the spine is loud. The pain is often radiated to the shoulders and upper limbs. The hands and fingers have numbness and electric shock, which can be aggravated by the neck activity.
Diagnosis
Differential diagnosis
1, osteomalacia: adult rickets. Mainly manifested as bone softening, bone-like tissue hyperplasia, bone deformation. Most of the causes are vitamin D and calcium and phosphorus deficiency. They are more common in women in cold and poor areas. A few cases are caused by renal tubular disease or enzyme deficiency, liver disease and anticonvulsant drugs. Early manifestations of lumbar pain, leg pain, inconvenient movement, bone tenderness, occasional convulsions or numbness, osteoporosis, skeletal deformities, fractures or pseudo-fractures or adult branch fractures, pelvic X-rays often trilobal Go to the mouth. The vertebral body is compressed into a wedge fracture or a double concave deformation. Nutritional factors can improve diet, supplement vitamin D and calcium, and increase activity. Other factors cause different treatments.
2, osteogenesis imperfects: Osteogenesis imperfecta (osteogenesis imperfecta) is a rare congenital skeletal development disorder, also known as fragile bone disease or brittle bone - blue sclera - deafness syndrome. It is characterized by fragile bone, blue sclera, deafness, and joint relaxation. It is a congenital hereditary pain caused by hypoplasia of mesenchymal tissue and collagen formation. The lesions are not limited to bones, but often involve other connective tissues such as eyes, ears, skin, teeth, and the like. The disease is hereditary and familial, but a few are single cases.
3, spondylitis: dysfunction caused by spinal deformity and rigidity, the patient's bending, chest expansion and neck flexion and other sports, will cause great pain and difficulty. In order to alleviate or prevent these adverse consequences, patients should learn self-care in addition to various examinations and treatments by doctors, such as careful and long-term positional exercise (initially receiving the guidance of a physiotherapist), in order to obtain and maintain the spine. The best position to enhance paravertebral muscle strength and increase lung capacity. The first thing to do is to maintain a proper position during rest. You should sleep on a hard bed and take the supine position to avoid the position of flexion deformity. Once the lesion has invaded the upper thoracic vertebrae and cervical vertebrae, the pillow should be stopped. Physical activity that causes persistent pain should be avoided: regular height measurement; maintaining a height record is a good measure to prevent early-onset spinal curvature. Chest wall lesions are more common, patients should be aware of the dangers of smoking, stop smoking.
