viral blepharitis
Introduction
Introduction to viral dermatitis Viral dermatitis is often caused by the whole body, and is classified into blister type, rash type and new type according to clinical manifestations. Common herpes simplex virus dermatitis and herpes zoster viral dermatitis: herpes simplex virus dermatitis: caused by herpes simplex virus, more complicated by systemic fever, can also be complicated by herpes simplex keratitis. Lesions can occur in the upper and lower jaws, and the following tendons are more common, which is consistent with the distribution of the underarms of the trigeminal nerve. The main indifference is treated with eye drops and eye ointment to keep the eyes clean and prevent secondary infections. Can't blink. Herpes zoster virus dermatitis: caused by herpes zoster virus infection of the trigeminal semilunar ganglion or the first branch of the trigeminal nerve, usually limited to one side. Before the onset, there are often too few pre-existing symptoms, such as general malaise, fever, and so on. Then there is severe neuralgia in the affected area. Then, in the affected side, the forehead skin and scalp are flushed and swollen, and clusters of transparent vesicles appear. Should take appropriate rest, improve the body's resistance, if necessary, give analgesics and sedatives, no need to use drugs when herpes is not broken. basic knowledge The proportion of illness: 0.002% Susceptible people: no specific people Mode of infection: non-infectious Complications: glaucoma Scleritis
Cause
Cause of viral dermatitis
Herpes simplex virus dermatitis is caused by herpes simplex virus-type I infection.
Herpes zoster viral dermatitis is caused by the varicella-zoster virus infecting the semilunar ganglion of the trigeminal nerve or the first branch of the trigeminal nerve.
Prevention
Viral dermatitis prevention
First, avoid jealousy: scratching can make the skin continue to suffer from mechanical stimulation and thicken, and even cause infection. Scratching also strengthens the effect, the patient becomes more and more itchy, the more itching and the more it catches, forming a vicious circle, and the course of the disease is prolonged.
Second, avoid hot water scalding: dermatitis, eczema in the acute phase, due to intradermal capillary vasodilation, there will be varying degrees of skin redness, papules, blisters. Wash or soak with hot water, increase the redness and swelling, increase the permeate, and aggravate the condition. Therefore, patients with dermatitis and eczema should use warm water to shower, avoid immersing in hot water and rubbing hard.
Third, avoid soap washing: especially alkaline soap, is a chemical irritant to the skin, can make dermatitis, eczema worse. If you need to use soap to decontaminate, it is best to choose a small irritating boric acid soap.
Fourth, avoid irritating food: pepper, wine, tea, coffee and other stimulating food, can make itching more likely to make eczema worse or relapse, should be contraindicated.
5. Avoid blind medication: dermatitis and eczema have a long course and are easy to repeat. Patients should cooperate with doctors for patient treatment. Some people treat eagerly, and do not apply a high concentration of antipruritic drugs to the skin lesions without medical treatment; instead, they aggravate the condition. Therefore, avoid using drugs without authorization.
Complication
Viral dermatitis complications Complications glaucoma scleritis
Herpes zoster virus dermatitis
Orbital herpes zoster often causes superficial keratitis, iridocyclitis, which is more likely when the nasal ciliary nerve is violated and herpes appears on the nose. Others can cause complications such as glaucoma, posterior scleritis and ophthalmoplegia.
Symptom
Viral ecdysis symptoms Common symptoms Herpes rash eyelid deformity blisters
Herpes simplex virus dermatitis
The lesion can invade the upper and lower iliac crest, but the following sputum is more common. The infected lesion can be confined to the sacral margin, or affect the periorbital skin, and is consistent with the distribution range of the trigeminal nerve. A cluster of translucent vesicles appear on the ankle skin with a tingling burning sensation. The initial blisters contain a clear yellow liquid that dries up in about a week, and the scars fall off without leaving scars, but may have mild pigmentation. A small number of cases showed the formation of erosive ulcers, characterized by the presence of erosion areas (1 to 4 mm) and skin ulcers (3 to 6 mm). According to the literature, up to 94% of patients have follicular conjunctivitis, and 15% develop chronic blepharitis. Lips and nasal vestibules can have the same damage, and severe cases have swollen lymph nodes.
Herpes zoster virus dermatitis
There are prodromal symptoms such as fever, chills, burnout and loss of appetite before the onset. Subsequent skin burning in the affected area, hypersensitivity and severe neuralgia. Then the skin is flushed, swollen, and clustered with miliary papules. After 48 to 72 hours, the skin erythema and maculopapular rash rapidly change into herpes. The lesion continues to develop for 3 to 5 days. 50% to 69% of patients have blepharospasm. The blister fluid starts to be transparent, and then turbid or merged into pustules, and the skin between the groups of blisters is normal. Herpes is confined to one side of the head, forehead, upper and lower skin, not over the midline of the face. Active infections often last 7 to 10 days. Followed by skin lesions. After 2 weeks, the blister scars fell off, and the lesion reached the dermis layer, leaving a permanent depressed scar and hyperpigmentation after healing. Double eyelashes, trichiasis, ptosis, and eyelid deformities can occur, preventing the eyelids from closing properly. After the inflammation subsides, the perception of the forehead, head, etc. is still diminished, and it takes several months to recover. Orbital herpes zoster often causes superficial keratitis, iridocyclitis, which is more likely when the nasal ciliary nerve is violated and herpes appears on the nose. Others can cause complications such as glaucoma, posterior scleritis and ophthalmoplegia.
Examine
Examination of viral dermatitis
Diseased base scrapers, often confirmed by multinucleated giant cells, Giemsa staining shows typical eosinophilic inclusion bodies. The positive rate of virus isolation in vesicles was 70%. Other specific tests include immunofluorescence electron microscopy, immunoperoxidase staining, radioimmunoassay, agar gel immunodiffusion, and DNA probes. Serological identification tests include enzyme-linked immunosorbent assay (ELISA), complement binding assay, immunoadhesive hemagglutination assay, and fluorescent antibody staining.
Diagnosis
Diagnosis and identification of viral dermatitis
diagnosis
Herpes simplex dermatitis can be diagnosed based on medical history and typical ocular manifestations.
Differential diagnosis
Diseased base scrapers, often confirmed by multinucleated giant cells, Giemsa staining shows typical eosinophilic inclusion bodies. The positive rate of virus isolation in vesicles was 70%. Other specific tests include immunofluorescence electron microscopy, immunoperoxidase staining, radioimmunoassay, agar gel immunodiffusion, and DNA probes. Serological identification tests include enzyme-linked immunosorbent assay (ELISA), complement binding assay, immunoadhesive hemagglutination assay, and fluorescent antibody staining. Comparison of serum virus antibody titers can identify primary and recurrent cases.
Herpes zoster viral dermatitis can be diagnosed based on medical history and clinical manifestations. Skin biopsy can be performed if necessary.
