myeloid hyperhidrosis
Introduction
Introduction to myeloid hyperhidrosis Medullary hyperhidrosis (medullaryhyperhidrosis) is often involved in taste receptors due to efferent stimuli. Therefore, myeloid hyperhidrosis, also known as gustatory sweating, can be divided into two types: physiological and pathological myelitis. basic knowledge The proportion of illness: 0.0002% Susceptible people: no special people Mode of infection: non-infectious Complications: depression
Cause
Cause of myeloid hyperhidrosis
(1) Causes of the disease
Medullary hyperhidrosis is caused by exudatory stimuli often involving taste receptors, causing localized sweating.
(two) pathogenesis
The pathogenesis is still not very clear. Normal efferent stimulation often involves taste receptors, and there are certain variations in the nucleus nucleus afferent and efferent arcs, resulting in different clinical manifestations.
Prevention
Myeloid hyperhidrosis prevention
The focus of our current prevention and treatment efforts should first focus on and improve those factors that are closely related to our lives, such as smoking cessation, proper diet, regular exercise, and weight loss. Appropriate exercise, enhance physical fitness and improve your disease resistance. Anyone who follows these simple and reasonable lifestyle knowledge can reduce the chance of suffering from hyperhidrosis.
Complication
Myeloid hyperhidrosis complications Complications depression
Hyperhidrosis can occur from a young age (about 6 years old) to adolescence, and the most troublesome patients are sweating in the palms, underarms, and soles of the feet. Young people are less likely to control their emotions, and are nervous, uneasy, shy, afraid, etc., making sweating more powerful, more anxious, and more sweating, causing a vicious circle. This may lead to the patient's unsociable, introverted, inferior and human interaction, lack of confidence in social situations of palm hyperhidrosis, and even inferiority, which will also affect learning and job hunting.
Symptom
Symptoms of myeloid hyperhidrosis Common symptoms Rogue pressure and orthostatic sweating, tears, neck taste hyperhidrosis
1. Physiological medullary nyperhidrosis
Many people suffer from limited sweating after eating spicy and fragrant foods and beverages. They are more common on the face, especially on the upper or both sides of the upper lip and cheek, as well as the scalp and knees, which usually appear within a few minutes. Often accompanied by vasodilation, it occurs in young people, with frequent hot climates and a family genetic predisposition.
2. pathologic medullary hyperhidrosis
Often involved in one side of the ear or under the ear, varying degrees, there are three clinical types: 1 localized trauma or disease caused by parotid gland; 2 caused by central nervous system diseases: such as syringomyelia or encephalitis; 3 thoracic sympathetic nerve Due to dry injury, the nucleus pulposus plays a role in all three types, but there are certain variations in the incoming and outgoing arcs, resulting in different clinical manifestations:
(1) auriculotemporal syndrome: also known as Krey's syndrome (Krey's syndrome) in the parotid or pre-auricular area surgery, trauma and cysts and other lesions after the injury of the ear nerves within 1 month to 5 years, diet, chewing When stimulating salivation, localized pain, vasodilation and sweating occur in the area of the deafness. The reason is that the auricular nerve and the parotid gland are involved at the same time. The parasympathetic fibers regenerated in the damaged parotid gland migrate to the distal end of the nerve, supporting the subarachnoid region. The sweat glands, tympanic plexus surgery, can eliminate the taste sweating of patients with this disease.
(2) chorda tympanic syndrome: due to the damage of the surrounding autonomic nerve fibers near the submandibular gland, the clinical manifestations are similar to deafness syndrome, occurring in the temporal and mandibular margin.
(3) Crocodile tear syndrome: is a lesion similar to taste hyperhidrosis, often occurs after facial nerve injury, the difference is that the patient has taste tears; originated from regulating tears and salivation Misleading or short-circuiting of the peripheral autonomic pathway.
(4) Sperm sweating caused by syringomyelia or encephalitis: may be caused by vagus and glossopharyngeal nerve stimulation, which may cause damage to the nucleus pulposus that controls sweating and salivation; clinical manifestations vary widely, and there is a wide range of outbreaks. Sweat reaction.
(5) medullary hyperhidrosis after thoracic sympathetic trunk injury: seen in patients with sympathectomy, lung cancer, spinal osteoma, subclavian aneurysm and thyroidectomy, because the sympathetic chain in the superior mediastinum is adjacent to the vagus nerve, in the sympathetic nerve After dry injury, the vagus nerve emits cholinergic fibers to the adjacent sympathetic trunk preganglionic fibers. Patients often experience sweating on the face, neck, trunk and upper limbs after eating or swallowing.
Examine
Examination of myeloid hyperhidrosis
1. Most of the head and limb imaging examinations are normal results, and secondary hyperhidrosis has performance related to the primary disease.
2. Autonomic nerve function test, which has auxiliary diagnosis and differential significance for hyperhidrosis.
Diagnosis
Diagnosis and differentiation of myeloid hyperhidrosis
diagnosis
Diagnosis can be based on the characteristics of clinical manifestations.
Differential diagnosis
Different from primary hyperhidrosis.
Primary hyperhidrosis: This type of hyperhidrosis is caused by hypersympathetic sympathetic rather than hyperhidrosis caused by disease. The vast majority of patients with hyperhidrosis belong to this type of hyperhidrosis. Although it does not affect the health of the body, it brings great inconvenience and trouble in life. At present, thoracic sympathetic surgery can be used to quickly cure such troubles.
