Quasena Forest Disease
Introduction
Introduction to Quassana Forest Disease The Quasina forest disease was recognized in 1957, when there was a fatal animal epidemic in the forested wild monkeys in the Chimago region of Kanataka, India, which was associated with disease outbreaks in villagers living on the edge of the forest. . Therefore, it is known as the "monkey disease", which was later named after the virus was isolated in the Quarsina. basic knowledge The proportion of illness: 0.005% Susceptible people: no specific population Mode of transmission: insect vector transmission Complications: massive hemoptysis
Cause
Cause of forest disease in Quasina
(1) Causes of the disease
The Quasina forest disease virus belongs to the genus Flavivirus of the genus Fagidae, and its antigenicity is related to other sputum virions, especially the virus of the Far Eastern sputum encephalitis, and the Omsk hemorrhagic fever virus.
(two) pathogenesis
The pathogenesis depends on vascular damage, thrombocytopenia, and the extent of brain, kidney, endocardium, myocardium, stomach, and intestinal bleeding. The virus mainly invades the blood vessels and nervous system, and the skin, mucous membranes, and visceral blood vessels are congested, and endothelial cells are damaged. Increased vascular permeability, tissue congestion, edema, cerebral edema can cause sensory changes, vasoconstriction can lead to collapse and shock, liver Kupffer cells can be seen hemosiderin deposition.
Prevention
Quasina forest disease prevention
The current preventive measures are mainly to control the source of infection and vaccination.
1. Control the source of infection
Mainly cockroach, anti-mite. In addition to reducing the chances of passing the virus to humans.
2, vaccination
Vaccination is an effective preventive measure.
Complication
Quasina forest disease complications Complications
Concurrent liver and kidney, functional damage and hematemesis, massive hemoptysis.
Symptom
Quasina forest disease symptoms Common symptoms Hemorrhagic tendency nausea fever vertigo vomiting diarrhea tremor muscle soreness lymph nodes swollen neck stiffness
Latent period of 3 to 8 days, sudden onset, fever, headache and severe muscle pain, some patients may have exhaustion, severe gastrointestinal disorders and bleeding, systemic lymphadenopathy, bronchial pneumonia, etc., some patients show two stages of disease process The second phase is characterized by meningeal encephalitis after no heat, manifested as severe headache, mental disorder, neck stiffness, severe tremor vertigo and abnormal nerve reflex.
Examine
Examination of the wildsana forest disease
Laboratory tests with Hemsk hemorrhagic fever.
ESR slows down.
Diagnosis
Diagnosis and identification of wildsana forest disease
diagnosis
There are fever, headache, muscle pain in the limbs, nausea, vomiting, diarrhea and bleeding tendency, oral mucosal rash, lymphadenopathy, etc., combined with the epidemic season and the source of the epidemic can be used for preliminary clinical diagnosis.
Differential diagnosis
The initial stage of fever should be differentiated from regression fever, typhoid fever, typhoid fever, leptospirosis, etc. The bleeding period should be differentiated from epidemic hemorrhagic fever and meningitis.
