Datura poisoning
Introduction
Introduction to Mandala Poisoning Daturastramonium (Daturastramonium) is also known as the golden flower. The whole plant is poisonous. It can cause poisoning by eating leaves, flowers, fruits and seeds. The seed contains the highest amount of poison. The children can take 3-8 seeds to take the mandala. Luo poisoning. The symptoms of poisoning vary with the amount of various alkaloids. The speed of onset varies with the amount of food intake and personal physique. Symptoms usually occur within 1/2 to 3 hours after eating the fruit. The initial symptoms are thirst. The skin and face are generally red, with red rash, dry and sweat-free, panic, irritability or lethargy. Afterwards, the hands and feet dance, stun, illusion, auditory hallucinations, severe convulsions, or tonicity, or clonic, often With moderate fever, a few can reach above 40 °C, the pupil dilated, the light reflection disappears or weakens, the blood pressure rises, the pulse speed is weak, after 12 to 24 hours, from the mania, stun into the coma, Death, if the rescue is timely or the poisoning is light, the above symptoms gradually disappear, and the pupil dilated usually returns to normal after 1 to 3 days, and even extended to 1 to 2 weeks, after which the patient completely forgets the condition. basic knowledge The proportion of illness: 0.0005% Susceptible people: no specific population Mode of infection: non-infectious Complications: coma
Cause
The cause of mandala poisoning
Poisonous plant food poisoning caused by ingestion of seeds, berries or seedlings of the genus Mandala. It has also been reported that drinking tea contaminated with mandala flowers has been poisoned.
Prevention
Mandala poisoning prevention
Most of the mandala poisoning is seen in preschool children. It is caused by ingestion. Therefore, it should be vigorously promoted that the mandala is poisonous and inedible, causing the attention of the masses to prevent accidents.
Complication
Mandala poisoning complications Complications
There are hallucinations, embarrassment, restlessness, tonicity or clonic convulsions, and finally coma, breathing superficial and other critical signs.
Symptom
Mandala poisoning symptoms common symptoms coma, cluminess, lethargy, tears, manicitis, ecchymosa, illusion, light reflection
The severity of the symptoms of poisoning may vary depending on the location of the edible plant. The speed of onset is also different due to the amount of food intake and personal physique. Symptoms usually occur within 0.5 to 3 hours after eating the fruit. Early symptoms are dry oropharyngeal, difficulty swallowing, dilated pupils, redness of skin and face, fever, irritability or lethargy. After 3-6 hours, the mental symptoms become more and more obvious, such as forgetfulness, hallucinations, auditory hallucinations, agitation, convulsions, disturbance of consciousness, dilated pupils, disappearance or weakening of light reflection, elevated blood pressure, and weak pulse rate. Severe cases often appear dizzy in 12 to 24 hours, slow breathing, blood pressure drops and even dangerous signs such as shock, coma and respiratory paralysis. If the rescue is timely or the poisoning is light, the above symptoms gradually disappear, and the dilated pupils usually return to normal after 1 to 3 days, and even extend to 1 to 2 weeks.
Examine
Mandala poisoning check
Qualitative analysis of urinary atropine: the urine is added with a little nitric acid on the water bath to dry, and then added with potassium hydroxide alcohol solution, if it is atropine, it is violet, and quickly turns red.
Blood routine examination: the total number of white blood cells, normal or slight increase, more than 60% of neutrophils.
Diagnosis
Diagnosis and diagnosis of mandala poisoning
Cases with unknown history of poisoning must be identified with hibernation, non-toxic or scarlet fever, encephalitis, meningitis, such as subcutaneous injection with methacholine, without sweating, tearing, increased saliva, gastrointestinal motility Enhance the normal response, suspected atropine poisoning, if necessary, the patient's urine can be used for qualitative analysis of atropine.
