choroiditis
Introduction
Introduction to choroiditis Choroiditis is a type of posterior uveitis. Because the choroidal blood vessels are derived from the short ciliary arteries, they can develop disease alone. However, because it is closely attached to the retina and supplies blood to the outer layer of the retina, it often involves the retina and is complicated by chorioretinitis. In the early stage of inflammation, the photoreceptor cells of the retina are stimulated, and there is a sense of flash in front of the eyes. When the inflammatory product enters the vitreous, it appears as vitreous opacity, and there are black spots floating in front of the eyes, and the visual acuity decreases to varying degrees. Under the ophthalmoscope, the spot in the vitreous can be found, the floc is suspended, and the direction of the eyeball is rotated. In the acute phase, there are yellow-white, patchy exudation, partial fusion, and micro-augmentation of the lesion. This is due to choroidal vasodilation and permeability. Reinforcement, caused by localized edema and cell infiltration, the retina of the lesion may also be affected. After the lesion is absorbed, the mild disease does not show traces. In severe cases, pigmentation or pigmentation occurs due to pigment epithelial lesions; choroid, small vessel layer Atrophy, large blood vessels are exposed; if the whole layer is atrophied, the sclera is exposed, forming a typical atrophy of black and white. basic knowledge The proportion of illness: 0.001% Susceptible people: no special people Mode of infection: non-infectious Complications: edema, retinal vasculitis, retinal detachment
Cause
Cause of choroiditis
The etiology of this disease has not been known to date. Most scholars believe that it is related to factors such as toxoplasmosis, histoplasmosis, amebic disease, piriformosis, toxoplasmosis, syphilis and tuberculosis.
The pathogenesis of this disease is believed to be caused by macular hemorrhage and exudation due to the invasion of new blood vessels originating from the subretinal choroid.
According to the extent of lesions: 1, diffuse choroiditis: (dffuse chorioiditis) for multiple exudative lesions fused into a piece, late showing large atrophy plaques, normal tissue between lesions. 2, disseminated choroiditis: (disseminated chorioiditis) scattered areas of the lesions, the size varies. 3, localized choroiditis: (circumseribed chorioiditis) lesions are limited to a certain part of the fundus, the macular area is more common.
Prevention
Choroiditis prevention
Early diagnosis, early treatment, prevention of inflammation. Proper rest, avoid fatigue, do regular work and rest, use your eyes for 5 to 10 minutes every hour for a long time, don't use your computer for too long, and you will have a better time when you feel unwell. In addition, you can take appropriate eye care products, and you can eat more foods containing vitamin C, such as vegetables and fruits.
(1) Pay attention to the impact of the climate, and avoid the cold and exogenous evils to induce the disease;
(2) quit smoking and alcohol;
(3) The diet should be light, avoiding the fat and spicy taste;
(4) Avoid excessive fatigue and mental stimulation.
Complication
Choroiditis complications Complications, retinal vasculitis, retinal detachment
1. The surface wrinkles of the macula: that is, the macular spot of the pre-retinal fibrous membrane is pearl-like.
2. Macular papilledema: Macular edema often causes cystic degeneration or even holes.
3. Retinal vasculitis: such as the toxoplasma to open the door around the arterial inflammation.
4. Retinal detachment: due to inflammation and exudation of vitreous mechanical traction.
Symptom
Symptoms of choroiditis Common symptoms Visually impaired sparks in front of the eyes or flashes in front of the eyes. Distortion of night blind vision is often foggy
1. Conscious symptoms:
Mainly due to vision loss and eye shine and black shadow flying, sometimes with small eyes and big eyes.
2. Signs:
(1) The vitreous is turbid, and the turbidity is composed of lymphocytes and fibrin, which are star-shaped, filamentous, and French-like, snowball-like.
(2) choroidal vasodilation, increased permeation, tissue infiltration edema, scattered diffuse grayish-white gray-yellow lesions in the fundus, round or oval, varying in size, blurred edges, mild elevation, single or multiple fusion . There is edema or occasional bleeding in the surrounding retina.
(3) After the acute phase, the lesion gradually turns white, forming a clear boundary with black patches in the middle and atrophy around the pigment. In this atrophic lesion, a hardened choroidal blood vessel is seen.
Examine
Choroiditis examination
Check the external eye is normal, under the ophthalmoscope can be seen in the vitreous point, floc suspension, reverse eye movement direction, acute yellow yellow, patchy exudation, partial fusion, lesion micro-up, due to choroid Vasodilatation, enhanced permeability, caused by localized edema and cell infiltration, the retina of the lesion may also be affected, after the lesion is absorbed, the mild cases do not show traces, and the severe cases have pigmentation or reproduction due to pigment epithelial lesions; choroid In the middle, the small blood vessel layer is atrophied and the large blood vessels are exposed; if the whole layer is atrophied, the sclera is exposed, forming a typical atrophy of black and white.
Diagnosis
Diagnosis and diagnosis of choroiditis
diagnosis
Diagnosis can be performed based on clinical performance and laboratory tests.
Differential diagnosis
Choroiditis should be differentiated from the middle uvitis, uveal inflammation in the posterior choroiditis, often retinal inflammation, and the central uvitis with ciliary body flat inflammation is often more obvious vitreous opacity.
