neuroma
Introduction
Introduction to neuroma Neurofibromas are also known as meningioma, neuroma, perineural fibroids, Schwann cell tumors, and perineural fibroblastoma. The disease has a wide range of names, reflecting different views on its source. It can be summarized in the following categories: the first category: neuroma or Schwannoma; the second category: neurofibromatosis or perihematoblastoma, It refers to connective tissue in which tumor cells are deepened from the inner mesoderm. Neurofibromas can originate from peripheral nerves, cranial nerves, and sympathetic nerves. The cutaneous neurofibroma is derived from the peripheral nerve and its interstitium, including the neuromembrane cells. Electron microscopy showed Schwann cell branching in collagen interstitial tissue. In coffee stains and clinically normal skin areas, large pigment particles are visible in the epidermal cells, which have never been seen in the normal skin of Albright syndrome. basic knowledge The proportion of sickness: 0.01% - 0.03% Susceptible people: no special people Mode of infection: non-infectious Complications: intracranial tumors epilepsy
Cause
Neuroma etiology
Cause:
The disease is also known as vonRecklinghausen disease, autosomal dominant inheritance, neuroectodermal abnormalities caused by distorted dominant genes, commonly incomplete and simple type. The cutaneous neurofibroma is derived from the peripheral nerve and its interstitium, including the neuromembrane cells. Electron microscopy showed Schwann cell branching in collagen interstitial tissue. In coffee stains and clinically normal skin areas, large pigment particles are visible in the epidermal cells, which have never been seen in the normal skin of Albright syndrome.
Prevention
Neuroma prevention
Early detection, early diagnosis, early treatment. A reasonable diet in life can consume more high-fiber and fresh vegetables and fruits, balanced nutrition, including essential nutrients such as protein, sugar, fat, vitamins, trace elements and dietary fiber, with a combination of vegetarian and vegetarian foods. Give full play to the complementary role of nutrients in food.
Complication
Neuroma complications Complications intracranial tumor epilepsy
The disease can worsen. As the tumor grows, depending on the location of the tumor, corresponding complications will occur.
Symptom
Symptoms of neuroma Common symptoms Allergic toe gaps numb limbs often have numbness, limbs, numbness, frontal mass, intermittent muscle contraction, limb numbness
1. Located in the limb mass, it is fusiform, and its nerves often have numbness, pain, and hyperesthesia on the far side of the inner limb.
2. Compression of the tumor can also cause numbness.
Examine
Neuroma examination
1. Because the tumor is mostly located in the limb, the armpit, it can also be located on the collarbone, neck and other parts. Therefore, when the patient is examined, it is necessary to touch, determine whether the tumor is easy to move or pedicle, whether the texture is hard, and observe the color of the tumor; and the tumor on the limb is all fusiform, and the limb of the nerve trunk is often distal. Numbness, pain, allergies and other symptoms.
2, need to take a pathological examination to confirm the diagnosis.
Diagnosis
Diagnosis and differentiation of neuroma
Diagnose based on
1. Most of the tumors are located in the limbs, armpits, and can also be located on the clavicle, neck and other parts.
2. The tumor on the limb is completely fusiform, and the limbs innervated by the nerve trunk often have numbness, pain, and hyperesthesia.
3. Compression of the tumor can also cause numbness.
Facial nerve tumors have different clinical manifestations due to their different growth sites. Early symptoms are concealed and clinically easily misdiagnosed. The clinical manifestations of the cerebellopontine angle appear to be similar to acoustic neuroma; it occurs in the humerus, and it appears to be Bell's palsy with sudden facial paralysis. The most common symptom of facial nerve sheath tumor is progressive facial nerve dysfunction, and facial muscle spasm can be the first symptom. It should be distinguished from primary facial muscle spasm. Primary facial muscle spasm is rarely accompanied by facial paralysis. Many scholars have suggested that tumors and other possibilities should be considered if conservative treatment is not improved for 16 to 20 weeks. Bell's palsy can also be ruled out if the facial paralysis is more than 6 to 12 weeks from the beginning to the full sputum. Sensorineural deafness, dizziness, and unilateral tinnitus may be clinical symptoms of the tumor located in the cerebellopontine angle and the internal auditory canal. For those with the above clinical symptoms, ear neurology, facial nerve electrophysiology test, and facial nerve function localization should be performed.
